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rabbit anti wnt9a  (Bioss)


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    Structured Review

    Bioss rabbit anti wnt9a
    Validation of key gene expression in cell models. (A) Phalloidin immunofluorescence staining of the HCM cell model established by ISO. (B) The integrated optical density (IOD) of phalloidin is significantly increased at 48 h. (C) ATRNL1 (mRNA) expression is significantly increased at 48 h. (D) <t>WNT9A</t> is significantly increased at 24 h and 48 h.
    Rabbit Anti Wnt9a, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rabbit+anti+wnt9a/WNT9A+Polyclonal+Antibody/pmc11424892-106-12-16
    Average 94 stars, based on 1 article reviews
    rabbit anti wnt9a - by Bioz Stars, 2026-09
    94/100 stars

    Images

    1) Product Images from "Identification of ATRNL1 and WNT9A as novel key genes and drug candidates in hypertrophic cardiomyopathy: integrative bioinformatics and experimental validation"

    Article Title: Identification of ATRNL1 and WNT9A as novel key genes and drug candidates in hypertrophic cardiomyopathy: integrative bioinformatics and experimental validation

    Journal: Frontiers in Molecular Biosciences

    doi: 10.3389/fmolb.2024.1458434

    Validation of key gene expression in cell models. (A) Phalloidin immunofluorescence staining of the HCM cell model established by ISO. (B) The integrated optical density (IOD) of phalloidin is significantly increased at 48 h. (C) ATRNL1 (mRNA) expression is significantly increased at 48 h. (D) WNT9A is significantly increased at 24 h and 48 h.
    Figure Legend Snippet: Validation of key gene expression in cell models. (A) Phalloidin immunofluorescence staining of the HCM cell model established by ISO. (B) The integrated optical density (IOD) of phalloidin is significantly increased at 48 h. (C) ATRNL1 (mRNA) expression is significantly increased at 48 h. (D) WNT9A is significantly increased at 24 h and 48 h.

    Techniques Used: Expressing, Immunofluorescence, Staining

    Pathological morphology and ATRNL1/WNT9A IHC staining in different groups. (A) HE stains of four groups: model, model + miR-1469, model + propylthiouracil, and model + triptolide. The model + miR-1469 and model + propylthiouracil groups showed a slight improvement of abnormalities in myocardial tissues. (B) IHC staining of ATRNL1 . (C) IHC staining of WNT9A . (D) Only propylthiouracil, but not miR-1469 or triptolide, significantly inhibits the expression of ATRNL1 in the HCM model. (E) All of the three drugs suppress WNT9A expression. * p < 0.05, ** p < 0.01, and *** p < 0.001.
    Figure Legend Snippet: Pathological morphology and ATRNL1/WNT9A IHC staining in different groups. (A) HE stains of four groups: model, model + miR-1469, model + propylthiouracil, and model + triptolide. The model + miR-1469 and model + propylthiouracil groups showed a slight improvement of abnormalities in myocardial tissues. (B) IHC staining of ATRNL1 . (C) IHC staining of WNT9A . (D) Only propylthiouracil, but not miR-1469 or triptolide, significantly inhibits the expression of ATRNL1 in the HCM model. (E) All of the three drugs suppress WNT9A expression. * p < 0.05, ** p < 0.01, and *** p < 0.001.

    Techniques Used: Immunohistochemistry, Expressing

    Related Articles

    Incubation:

    Article Title: Identification of ATRNL1 and WNT9A as novel key genes and drug candidates in hypertrophic cardiomyopathy: integrative bioinformatics and experimental validation
    Article Snippet: Then, sections were treated with citrate buffer and freshly prepared in 3% hydrogen peroxide, followed by sealing with bovine serum albumin (BSA) (Solarbio) for 30 min at 37°C. .. The sections were incubated with rabbit anti- ATRNL1 (bs-11504R, Bioss, 1/200) or rabbit anti- WNT9A (bs-1933R, Bioss, 1/200) overnight at 4°C. .. After washing, horseradish enzyme-labeled goat anti-rabbit IgG (H + L) (ZB-2301, Nakasugi Jinqiao, 1/100) was added dropwise and incubated at 37°C for 30 min. After re-staining with hematoxylin, the slices were blocked and observed under the microscope (CX43, OLYMPUS).



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    Validation of key gene expression in cell models. (A) Phalloidin immunofluorescence staining of the HCM cell model established by ISO. (B) The integrated optical density (IOD) of phalloidin is significantly increased at 48 h. (C) ATRNL1 (mRNA) expression is significantly increased at 48 h. (D) <t>WNT9A</t> is significantly increased at 24 h and 48 h.
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    Validation of key gene expression in cell models. (A) Phalloidin immunofluorescence staining of the HCM cell model established by ISO. (B) The integrated optical density (IOD) of phalloidin is significantly increased at 48 h. (C) ATRNL1 (mRNA) expression is significantly increased at 48 h. (D) <t>WNT9A</t> is significantly increased at 24 h and 48 h.
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    Image Search Results


    Validation of key gene expression in cell models. (A) Phalloidin immunofluorescence staining of the HCM cell model established by ISO. (B) The integrated optical density (IOD) of phalloidin is significantly increased at 48 h. (C) ATRNL1 (mRNA) expression is significantly increased at 48 h. (D) WNT9A is significantly increased at 24 h and 48 h.

    Journal: Frontiers in Molecular Biosciences

    Article Title: Identification of ATRNL1 and WNT9A as novel key genes and drug candidates in hypertrophic cardiomyopathy: integrative bioinformatics and experimental validation

    doi: 10.3389/fmolb.2024.1458434

    Figure Lengend Snippet: Validation of key gene expression in cell models. (A) Phalloidin immunofluorescence staining of the HCM cell model established by ISO. (B) The integrated optical density (IOD) of phalloidin is significantly increased at 48 h. (C) ATRNL1 (mRNA) expression is significantly increased at 48 h. (D) WNT9A is significantly increased at 24 h and 48 h.

    Article Snippet: The sections were incubated with rabbit anti- ATRNL1 (bs-11504R, Bioss, 1/200) or rabbit anti- WNT9A (bs-1933R, Bioss, 1/200) overnight at 4°C.

    Techniques: Expressing, Immunofluorescence, Staining

    Pathological morphology and ATRNL1/WNT9A IHC staining in different groups. (A) HE stains of four groups: model, model + miR-1469, model + propylthiouracil, and model + triptolide. The model + miR-1469 and model + propylthiouracil groups showed a slight improvement of abnormalities in myocardial tissues. (B) IHC staining of ATRNL1 . (C) IHC staining of WNT9A . (D) Only propylthiouracil, but not miR-1469 or triptolide, significantly inhibits the expression of ATRNL1 in the HCM model. (E) All of the three drugs suppress WNT9A expression. * p < 0.05, ** p < 0.01, and *** p < 0.001.

    Journal: Frontiers in Molecular Biosciences

    Article Title: Identification of ATRNL1 and WNT9A as novel key genes and drug candidates in hypertrophic cardiomyopathy: integrative bioinformatics and experimental validation

    doi: 10.3389/fmolb.2024.1458434

    Figure Lengend Snippet: Pathological morphology and ATRNL1/WNT9A IHC staining in different groups. (A) HE stains of four groups: model, model + miR-1469, model + propylthiouracil, and model + triptolide. The model + miR-1469 and model + propylthiouracil groups showed a slight improvement of abnormalities in myocardial tissues. (B) IHC staining of ATRNL1 . (C) IHC staining of WNT9A . (D) Only propylthiouracil, but not miR-1469 or triptolide, significantly inhibits the expression of ATRNL1 in the HCM model. (E) All of the three drugs suppress WNT9A expression. * p < 0.05, ** p < 0.01, and *** p < 0.001.

    Article Snippet: The sections were incubated with rabbit anti- ATRNL1 (bs-11504R, Bioss, 1/200) or rabbit anti- WNT9A (bs-1933R, Bioss, 1/200) overnight at 4°C.

    Techniques: Immunohistochemistry, Expressing